Yes those 3 would be major ones based on the historical literature but we are now learning about dht being cardiotoxic itself, steroids hitting and growing the heart via ar and mr, thick blood itself raising the loads and all mixing to generate fibrosis.
Classical methods like ekg, echo, holtor, bloods and urinalysis won't exactly pick up on fibrosis and scarring.
The reason the auxiliary drug stack increases in size unfortunately is because we are simply finding new pathways we may have only had theory or mechanism to suggest.
Its really not ideal and honestly is off putting to myself but i think as we continue to progress our understanding of our physiology anabolics will be approached even more carefully than now.
It was a cool experiment but the process has actually reduced the total compounds im willing to run further.
Prior i hadn't touched most fat burners or stims and left a few anabolics on the table. Hadn't touched 99% of peptides or sarms. Now deff leaving more on the table a few more anabolics lol.